- Global cancer incidence is increasing, particularly among those under 50, with a notable rise in breast, uterine, prostate, lung, colorectal, pancreatic, and stomach cancers, with early-onset breast cancer incidence climbing by 3.8% annually between 2016 and 2019
- The global death toll from early-onset cancers has surged by nearly 28% from 1990 to 2019, with predictions indicating a potential rise in mortality rates
- Dietary changes, particularly the increase in obesity and consumption of processed foods rich in linoleic acid (LA), a polyunsaturated fat found in seed oils, are identified as major contributors to the rising cancer rates
- LA intake is linked to several mechanisms promoting cancer, including disruption in energy production in cells, hormonal imbalances, increased oxidative stress, and damage to cellular and mitochondrial membranes
- Limit your LA intake and avoid processed foods and certain animal products to mitigate your cancer risk
(Mercola)—According to the latest statistics, global cancer incidence is on the rise, especially among younger people. Between 1990 and 2019, incidence of 29 cancers rose worldwide in those younger than 50, with rates rising faster among women. Cancers on the rise include breast, uterine, prostate, lung, colorectal, pancreatic and stomach cancers.
Between 2016 and 2019, early-onset breast cancer incidence rose by 3.8% annually.1 As reported by the journal Nature in mid-March 2024,2 even teens are now being diagnosed with “improbable” cancers previously only seen in seniors, such as advanced gastrointestinal cancers. And it’s not just happening in the U.S. Oncologists in China and India have also noted this trend.3 According to Nature:4
“Statistics from around the world are now clear: the rates of more than a dozen cancers are increasing among adults under the age of 50. This rise varies from country to country and cancer to cancer, but models based on global data predict that the number of early-onset cancer cases will increase by around 30% between 2019 and 2030.5
In the United States, colorectal cancer — which typically strikes men in their mid-60s or older — has become the leading cause of cancer death among men under 50.6 In young women, it has become the second leading cause of cancer death …
[The] number of deaths from early-onset cancers has risen by nearly 28% between 1990 and 2019 worldwide. Models also suggest that mortality could climb …
As calls mount for better screening, awareness and treatments, investigators are scrambling to explain why rates are increasing. The most likely contributors — such as rising rates of obesity and early-cancer screening — do not fully account for the increase.”
Likely Culprits
The conventional medical model is beyond clueless when they believe the solution for this dilemma is better screening. This is unadulterated nonsense and fails to address the cause, which is a disruption in the microbiome that obliterates the healthy obligate anaerobes in the large intestine.
When your body fails to make enough cellular energy because the mitochondria are dysfunctional as a result of being regularly poisoned by LA and estrogen, they simply are unable to maintain an environment that allows the healthy beneficial bacteria to grow.
When the beneficial bacteria disappear, they are replaced by facultative anaerobes that tolerate oxygen, as a result of the lack of competitive inhibition by the beneficial bacteria. It is these bacteria that are increasing GI cancers, not a lack of screening.
Excessive LA Intake Promotes Cancer
The Nature article7 does, however, point to there being a dietary culprit, stating “The prominence of gastrointestinal cancers and the coincidence with dietary changes in many countries point to the rising rates of obesity and diets rich in processed foods as likely culprits in contributing to rising case rates.”
While the Nature article does not go into specifics about what it might be about our modern diets that contribute to cancer, my own research has singled out linoleic acid (LA) — an omega-6 fat found in seed oils (and hence most processed foods) — as a key contributor.
Like other experts in bioenergetic medicine, I’m convinced that chronic diseases such as Type 2 diabetes, Alzheimer’s and cancer are bioenergetic diseases rooted in dysfunctional metabolism resulting in poor energy production, and polyunsaturated fats (PUFAs) appear to be a primary contributing factor in this chain of events. As reported by bioenergetic researcher Georgi Dinkov in an October 2022 blog post:8
“The bad news for PUFA (in this case linoleic acid) just keep piling on. At this point, there is hardly a chronic condition out there that has not been linked to elevated PUFA and/or their peroxidation products.
[A December 2022 study in Antioxidants & Redox Signaling9] makes the argument that PUFA peroxidation byproducts (created by the increased reactive oxygen species (ROS) itself driven by PUFA) such as 4-hydroxynonenal (4-HNE), are the cause of diabetes and cancer.
In fact, the study makes the argument that the two conditions are basically slightly different points on the same spectrum of systemic disease (i.e. cancer being the more severe one).”
The study10 published in Antioxidants & Redox Signaling highlights the increased risk diabetic patients face in developing several types of cancer, including hepatocellular carcinoma, pancreatic cancer, colorectal cancer, and breast cancer.
LA intake is linked to several mechanisms promoting cancer, including disruption in energy production in cells, hormonal imbalances, increased oxidative stress, and damage to cellular and mitochondrial membranes.
This elevated risk is attributed to several factors that promote cancer growth in diabetic individuals. These factors include insulin resistance, hyperglycemia, dyslipidemia, chronic inflammation, and elevated levels of insulin-like growth factor-1 (IGF-1). These conditions contribute to reductive stress that disrupt the body’s redox balance. As explained by the authors:11
“The consequent oxidative stress associated with lipid peroxidation appears to be a possible pathogenic link between cancer and diabetes” and “the major bioactive product of oxidative degradation of polyunsaturated fatty acids (PUFAs), the reactive aldehyde 4-hydroxynonenal (4-HNE) … may be the key pathogenic factor linking diabetes and cancer …
Controlling the production of 4-HNE to avoid its cytotoxicity to normal but not cancer cells while preventing its diabetogenic activities could be an important aspect of modern integrative biomedicine.”
4-HNE Linked to Obesity and Cancer
In her book, “Deep Nutrition: Why Your Genes Need Traditional Food,”12 Dr. Cate Shanahan details the hazards of 4-HNE, which forms during the processing of most vegetable oils. 4-HNE is highly toxic to gut bacteria, and consumption of 4-HNE has been correlated with having an obesogenic balance of gut flora.
4-HNE is also cytotoxic (toxic to cells), causes DNA damage, and instigates free radical cascades that damage the mitochondrial membrane. As noted by Shanahan in a 2017 interview:
“You can’t design a better delivery vehicle for a toxin that’s going to destroy your health slowly over the course of maybe 10, 20 years, depending on the genetics of your antioxidant system capacity.”
Importantly, 4-HNE occurs even if the oil is obtained from organic crops. It’s an intrinsic byproduct of the refining and processing of the oil, no matter how healthy the oil initially was.
Elevated LA Destroys Mitochondrial Function
Dinkov’s blog also draws attention to another noteworthy study,13 albeit its focus is on Alzheimer’s Disease instead of cancer. The study reveals that individuals with Alzheimer’s have cells that are inefficient at producing energy.
This inefficiency stems from a deficiency in glycocholic acid and an excess of LA. Essentially, LA triggers the production of damaging reactive oxygen species (ROS), which then hampers the cells’ ability to generate energy.
Moreover, PUFAs, like LA, disrupt your body’s hormonal equilibrium, imitating the effects of estrogen and cortisol while counteracting androgens and progesterone. Additionally, they alter cellular interactions with water, making cells more water-attracting. Dinkov suggests that these effects, beyond merely generating ROS, play a much more critical role in the harmful effects of PUFAs on a wide variety of chronic diseases, including cancer.
Seed Oils Undermine Your Health in Myriad Ways
In summary, seed oils in general and LA in particular have been shown to harm health by:14
- Make your fat cells more insulin sensitive, thereby causing insulin resistance.15
- Creating high amounts of oxidation products when used in cooking (as they are very susceptible to heat), including aldehydes, which are what cause oxidized low-density lipoprotein (LDL) associated with heart disease. Aldehydes also crosslink tau protein and create neurofibrillary tangles, thereby contributing to the development of neurodegenerative diseases.
- Damaging the endothelium (the cells lining your blood vessels) and causing an increase in penetration of LDL and very low-density lipoprotein (VLDL) particles into the subendothelium.16
- In other words, these oils get integrated in your cell and mitochondrial membranes, and once these membranes are damaged, it sets the stage for all sorts of health problems. With a half-life of 600 to 680 days,17 it can take years to clear them out of your body. They also get incorporated into tissues such as your heart and brain.
- Damaging your mitochondria and DNA by making your cell membranes more permeable, allowing things to enter that shouldn’t.
- Making the cell membrane less fluid, which impacts hormone transporters in the cell membrane and slows your metabolic rate.
- Inhibiting cardiolipin,18 an important component of the inner membrane of your mitochondria that needs to be saturated in DHA to perform optimally and facilitate optimal function of the electron transport chain and production of ATP. Cardiolipin can be likened to a cellular alarm system that triggers apoptosis (cell death) by signaling caspase-3 when something goes wrong with the cell.
- If the cardiolipin is not saturated with DHA, it cannot signal caspase-3, and hence apoptosis does not occur. As a result, dysfunctional cells are allowed to continue to grow, which can turn into a cancerous cell.
- Inhibiting the removal of senescent cells, i.e., aged, damaged or crippled cells that have lost the ability to reproduce and produce inflammatory cytokines that rapidly accelerate disease and aging.
- Stripping your liver of glutathione (which produces antioxidant enzymes), thereby lowering your antioxidant defenses.19
- Inhibiting delta-6 desaturase (delta-6), an enzyme involved in the conversion of short-chained omega-3s to longer chained omega-3s in your liver.20
- Exposing you to toxic 4-HNE, which is highly toxic to gut bacteria, causes DNA damage, and instigates free radical cascades that damage your mitochondrial membranes.21
- Exposing you to glyphosate residues, as most vegetable oils are made with genetically engineered crops. Glyphosate has been shown to disrupt the tight junctions in your gut and increase penetration of foreign invaders, especially heated proteins, which can cause allergies.
How to Avoid These Dangerous Fats
Considering the profoundly serious damage they cause, eliminating seed oils from your diet can go a long way toward improving your health. This includes soy, canola, sunflower, grapeseed, corn, safflower, peanut and rice bran oil.
Also, be mindful of olive oil and avocado oil, as both are commonly adulterated with cheaper seed oils. That said, even pure olive and avocado oil are loaded with LA. If, like me, you’re in the habit of eating olive oil, I would strongly encourage you to limit your intake to 1 tablespoon per day or less. In my view, olive oil is not a magic bullet and if you are already consuming 80 grams of LA per day, it will only worsen, not help, your health.
Additionally, as you can see in the video below from Brad Marshall, whose interview with me will post shortly, olive oil is no all it is cracked up to be.
To avoid these oils, don’t cook with them, of course, but also avoid processed foods, condiments, fast foods and restaurant foods. If you eat out, you’re undoubtedly eating unhealthy amounts of seed oils, as most restaurant foods are loaded with it.
Fried foods, dressing and sauces tend to be key culprits. Your best bet is to prepare most of your food at home, so you know what you are eating and, in the case of seed oils, what you’re not. Chicken and pork are also high in LA and are therefore best avoided. Since these animals, even healthy organically grown animals, are typically fed grains, they are loaded with omega-6 fats and may have 10 times the LA content that beef, lamb or buffalo do.
How Much Linoleic Acid Is Too Much?
Ideally, consider cutting LA down to below 5 grams per day, which is close to what our ancestors used to get before all of these chronic health conditions, including obesity, diabetes, heart disease and cancer, became widespread. If olive oil puts you over the limit, consider cooking with tallow or lard instead.
If you’re not sure how much you’re eating, enter your food intake into Cronometer — a free online nutrition tracker — and it will provide you with your total LA intake. The key to accurate entry is to carefully weigh your food with a digital kitchen scale so you can enter the weight of your food to the nearest gram.
Cronometer will tell you how much omega-6 you’re getting from your food down to the 10th of a gram, and you can assume 90% of that is LA. Anything over 10 grams is likely to cause problems. To learn more about the biological impact of LA, be sure to watch the video at the top of this article, and/or read through my in-depth report, “Linoleic Acid — The Most Destructive Ingredient in Your Diet.”
mRNA Injections and Turbo Cancer
Another significant factor in the increase of cancer cases is likely the COVID vaccine. I’m convinced that excessive intake of LA was already a key issue contributing to rising cancer rates before the COVID era. Nonetheless, the introduction of mRNA vaccines has compounded the problem.
Specifically, these vaccines seem to be linked to the emergence of “turbo cancers,” which are rapidly growing cancers that often prove fatal. The sheer speed of their growth leaves little opportunity for effective treatment.
Canadian oncologist and cancer researcher Dr. William Makis in the HighWire interview above,22 replacing the uracil in the RNA used in the COVID shots with synthetic methylpseudouridine (to improve RNA stability) is known to be problematic.23 When substituting parts of the code in this way, the resulting protein can easily get misfolded, and this has been linked to a variety of chronic diseases.24
Importantly, the insertion of synthetic pseudouridine can suppress your innate immune surveillance by dampening the activity of toll-like receptors, and one downstream effect of that is reduced cancer surveillance.
“The more mRNA shots you take, the greater the immune system damage, the greater your risk of impaired cancer surveillance and hence, the greater your risk of turbo cancer,” Makis says.
The World Health Organization’s International Agency for Research on Cancer (IARC) predicts more than 35 million new cancer cases in 2050 — a 77% increase from the estimated 20 million cancer cases that occurred in 2022.25 Not surprisingly, the same drug companies that made the mRNA shots are now in a race to be the first to come up with new cancer drugs, including Pfizer and Johnson & Johnson.26
mRNA-Based Cancer Drugs Under Development
Disturbingly, many of the cancer drugs currently under development depend on the same mRNA-based technology responsible for the turbo cancer trend in the first place. Here are a handful of notable examples:
- Personalized cancer vaccines — Companies like BioNTech (which partnered with Pfizer for the COVID-19 vaccine) and Moderna have been working on personalized mRNA vaccines for cancer. These vaccines are designed to stimulate the patient’s immune system to target tumors based on the unique mutations in their cancer cells.27
- mRNA technology for solid tumors — Moderna is also developing an mRNA vaccine targeting solid tumors.28 This approach involves using mRNA to produce tumor-specific antigens inside the body to encourage the immune system recognize and destroy cancer cells.
- Combination therapies — Researchers are exploring the combination of mRNA vaccines with existing cancer treatments, such as checkpoint inhibitors, to enhance the immune system’s ability to fight cancer.
- Neoantigen targeting — Several biotech firms are focusing on neoantigens, which are new antigens that arise due to tumor mutations.29
- mRNA-based CAR-T cell therapy — Efforts are also being made to combine mRNA technology with CAR-T cell therapy, a type of treatment that modifies a patient’s T-cells to attack cancer cells. mRNA is used to allow CAR-T therapy to be rapidly tailored to different types of cancer.30
Considering the mRNA technology itself appears to produce carcinogenic results, I’d be extremely wary about cancer drugs based on it. Time will tell if they can somehow perfect it, but why wait? As mentioned, excess LA in the diet is a major driver of cancer, with or without the mRNA shots, and that is something you have complete control over.
As discussed in previous articles, you also want to avoid excess estrogen, which is a major driver of many cancers, especially breast cancer. For a refresher, see “What You Need to Know about Estrogen and Serotonin.”
- 1 JAMA Netw. Open 2024; 7: e2353331
- 2, 3, 4, 7 Nature March 13, 2024
- 5 BMJ Oncol. 2023; 2: e000049 (2023)
- 6 CA Cancer J. Clin. 2024; 74: 12–49
- 8 Haidut.me October 26, 2022 (Archived)
- 9, 10, 11 Antioxidants & Redox Signaling December 2022;37(16-18):1222-1233
- 12, 19, 20, 21 Deep Nutrition: Why Your Genes Need Traditional Food by Dr. Cate Shanahan
- 13 Front Aging Neurosci September 23, 2022; 14
- 14 Spotify Joe Rogan Podcast #1551 with Paul Saladino
- 15 YouTube June 23, 2020
- 16 Open Heart 2018;5:e000898
- 17 Journal of Lipid Research 1966 Jan;7(1):103-11
- 18 YouTube, Omega-6 Apocalypse 2, Chris Knobbe August 25, 2021
- 22 Rumble, The HighWire with Del Bigtree, Is the COVID Vaccine Causing Turbo Cancers? September 22, 2023
- 23 International Journal of Vaccine Theory, Practice and Research May 10, 2021; 2(1): 402-444
- 24 Nature Medicine December 6, 2011; 17: 1536-1538
- 25 World Health Organization February 1, 2024
- 26 MakisMD Substack March 12, 2024
- 27 Cancer.gov January 20, 2022
- 28 Moderna Clinical Trial of Cancer Treatment for Adults with Solid Tumors
- 29 Genome Medicine January 25, 2024;16(1):17
- 30 Penn Medicine News Release January 6, 2022
Two Storms, One Harvest
Every food crisis in living memory has been a one-shock event. The 2008 price spike was a commodity bubble. The 2020 shortages were a logistics failure. The 2022 grain scare was a war on one exporter’s ports. Each time, the system bent, adjusted, and recovered, and each time the experts assured us afterward that global markets are simply too big and too diversified to fail.
What nobody in Washington seems eager to discuss is that 2026 is shaping up to be something the modern food system has never actually faced. Two independent shocks, one climatic and one geopolitical, are converging on the same harvest cycle at the same time. Not sequentially. Simultaneously.
Start with the weather. The Pacific Ocean is currently building toward what forecasters now openly call a record event. NOAA’s Climate Prediction Center puts the odds of at least a strong El Niño near 88 percent, with roughly two in three odds it reaches “very strong” status, the tier reserved for perhaps three or four events in the entire satellite era. Every major global model now projects a median peak in Super El Niño territory, and most of them project it exceeding the 2015-16 event, which until now held the modern record. Sea surface anomalies were already brushing the super threshold in mid-July, months before these events normally peak. The atmosphere has already shifted into El Niño mode, and the event is forecast to crest in late fall and early winter.
This is not about “climate change.” It’s about the standard cycles of weather, and the cycle we’re currently in is one that has likely devastated societies in the past. We’re better prepared as a society today, but not all Americans are equally prepared.
Serious households have started doing the quiet math on their own. Grocery bills tell part of the story, and the forecast maps tell the rest, which is why long-term food storage has moved from fringe hobby to mainstream line item in the family budget, with established suppliers like Heaven’s Harvest seeing demand from people who five years ago would have rolled their eyes at the idea. That instinct is not paranoia. It is pattern recognition, and the pattern is worth walking through carefully.
Editor’s Note: Heaven’s Harvest IS a sponsor, but the warnings of this article are real and would be written even if we didn’t have a survival food sponsor. With that said, those who take advantage of what they offer can use promo code “Patriot” for 15% off.
The Fertilizer Clock Is Already Running
While the Pacific warms, the second shock has been unfolding in the Strait of Hormuz. The conflict with Iran turned the world’s most important energy chokepoint into a contested waterway, and the consequences reach far beyond the gas pump. Roughly a third of global fertilizer trade moves through Hormuz, and the disruption sent urea prices up 86 percent year over year by March, with a 53 percent jump in a single month.
The World Bank projects energy prices rising about 24 percent in 2026 and fertilizer about 31 percent. By its own accounting, fertilizer prices ran 35 percent higher in the first five months of this year than the same period last year.
Here is the mechanism the nightly news will not explain. Fertilizer is not a grocery item. It is a time-delayed input. The nitrogen a farmer in Iowa or Punjab could not afford to apply this spring does not show up as a problem this spring. It shows up as a thinner harvest six to twelve months later.
The World Bank’s own food security brief concedes that the effects of reduced applications earlier this season “are likely to become visible only later in harvest outcomes.” Translate that from institutional language into plain English and it means this. The damage is already done, it is already in the ground, and we are simply waiting for it to arrive on the shelf.
Now check the calendar. Six to twelve months from the spring planting season lands us squarely in late 2026 and early 2027. Which is precisely when the strongest El Niño in the instrumental record is forecast to peak, bringing its signature droughts to Southeast Asia, Australia, southern Africa, northern Brazil, and South Asia, the very regions that grow the world’s rice, sugar, and oilseeds.
The World Bank warns openly that a strong El Niño “could disrupt multiple crop belts simultaneously” on top of the conflict-driven input costs. Their baseline projection assumes the Middle East disruptions ease by autumn. What in the last two years of Middle East history suggests that assumption is safe?
The System Has No Slack Left
The comfortable answer is that global markets always adjust. But adjustment requires slack, and the slack is gone. Global cereal production is expected to decline from last year’s records even before El Niño does its work. The UN World Food Programme, hardly a den of right-wing preppers, is calling this the most significant disruption to its supply chains since Covid and the invasion of Ukraine, and its supply chain director put the stakes bluntly.
Today’s supply chain challenges are tomorrow’s hunger crisis.
There is also a political dimension that markets cannot price. When food gets scarce, governments do not behave like economists. They behave like politicians. Export bans, hoarding mandates, and panic buying at the national level turned the modest rice shortfall of 2008 into a global crisis, and analysts are already warning that import-dependent nations are the first dominoes.
The 2015-16 Super El Niño, a far weaker event than what is now forecast, threw tens of millions into food stress across Africa and Asia. This one is projected to be stronger, and it arrives with fertilizer already rationed by price and shipping lanes already contested by missiles.
What Joseph Knew
Scripture does not treat preparation for lean years as faithlessness. It treats it as wisdom delivered in advance to those willing to act on it.
Behold, there come seven years of great plenty throughout all the land of Egypt: And there shall arise after them seven years of famine; and all the plenty shall be forgotten in the land of Egypt.
Joseph did not respond to that warning with a hashtag or a committee. He stored grain during the years of abundance, and when the famine came, Egypt stood while its neighbors begged. The lesson is not that famine is certain. It is that the time to prepare is precisely when preparation still looks optional.
Nobody who filled a pantry in a year of plenty has ever regretted it, and nobody standing in an empty aisle has ever been glad he waited for certainty.
None of this calls for panic, and panic is the enemy of sound judgment anyway. It calls for the same unglamorous prudence our grandparents considered ordinary. Keep some cash margin, know your local growers, and put real food in deep storage while it is cheap and available, because the entire arc of this story is that cheap and available is a closing window.
Families looking for a straightforward place to start can visit Heaven’s Harvest and use promo code Patriot for 15 percent off long-term storable food. The forecasts may yet soften, the strait may yet reopen, and we should pray they do. But hope is a fine thing to hold and a foolish thing to eat.
Eat meat! We are carnivores…and consider the globalists don’t want us to eat meat eggs butter bacon. All good health promoting food. Instead, they want you to eat carbs which turn to sugar, sugars in many forms and then add secret ingredients which encourage addictions to their junk foods.
Pretty simeple. Eat a species specifice food. In our case red meat…Beef…exactly what Bill Gates doesn’t want you to eat.
Me? I’m eating meat….f the bugs and their promoters.